Metabolic & endocrine
Desmopressin
A V2-selective vasopressin analogue approved in nasal, oral and injectable formulations — each tied to its own specific indications.
Also known as: DDAVP, desmopressin acetate, Stimate, Nocdurna
Evidence passport
Last reviewed 2026-08-23
- Evidence level
- L5Practice-changing
- Confidence
- High confidence
- Human efficacy
- 85/100
- Human safety
- 80/100
- Regulatory status
- Approved medicine
- Development
- Approved
- Routes
- NasalOralIVSC injection
- Evidence base
- Includes human data
- Furthest stage
- Approved
- Source curation
- Sources fully curated
- Clinical maturity
- Long-established approved medicine family (multiple formulations)
Overview
Clinical maturity: Long-established approved medicine family (multiple formulations)
Routes & formulations
Studied / approved routes
Nasal, oral and injectable desmopressin products exist with different approved indications; a formulation approved for one use is not evidence for another. The haemostatic (clotting-factor-releasing) use belongs to specific injectable and high-concentration nasal products, not to every desmopressin formulation.
Route availability is not evidence of efficacy, and evidence from one route does not transfer to another. The Registry never provides dosing or sourcing information.
Peptide lifecycle
Discovery
Preclinical
Phase I
Phase II
Phase III
Approved
What it is
A synthetic analogue of the natural antidiuretic hormone vasopressin, modified to act selectively on V2 receptors — reducing urine output and, via a separate endothelial effect, raising certain clotting factors.
What people claim
- Controls excessive urination in central diabetes insipidus
- Reduces night-time urine production in specific labelled indications
- Supports haemostasis in certain mild bleeding disorders
What the evidence actually says
One of the clearest demonstrations that peptide medicines can work by nasal, oral and injectable routes — but each formulation was approved on its own evidence, for its own indications. Hyponatraemia (low blood sodium) is the key labelled risk and the reason fluid-management advice sits in its labelling.
Human evidence
- Decades of controlled trials and post-marketing use across approved indications.
- Randomised trials support specific nocturia and central diabetes insipidus indications for specific products.
Preclinical evidence
- Receptor pharmacology establishing V2 selectivity over V1 pressor effects.
Animal and cell findings are Level 1–2 evidence.
Mechanism and pathways
- V2-receptor agonism in the kidney collecting duct increases water reabsorption.
- Endothelial V2 stimulation releases von Willebrand factor and factor VIII, underpinning labelled haemostatic uses.
Safety and unknowns
- Hyponatraemia, sometimes severe, is the central labelled risk; fluid advice accompanies treatment.
- Formulation and indication matter: contraindications and monitoring differ between products.
Cancer relevance
No concern identified
Regulatory status
Approved in the US and many other markets, but approvals are formulation- and indication-specific: for example products for central diabetes insipidus and certain nocturia indications, and specific injectable and high-concentration nasal products for haemostatic use in certain bleeding disorders. No single product covers every indication.
Evidence grade
Replicated large randomised trials, pivotal Phase 3 programmes, or a major regulatory decision.
What would change our rating?
- Not applicable at this evidence tier.
References and sources
- Regulatory / labelFDA label: DDAVP (desmopressin acetate) injection and rhinal tube
- Regulatory / labelFDA Orphan Drug Designations and Approvals: desmopressin listing
Source hierarchy: regulatory documents and trial registries first, peer-reviewed work next, sponsor material flagged as non-independent. We never use supplier or clinic marketing as evidence, and where a verified source has not yet been curated we say so rather than inventing one.
Evidence change history
No recorded changes yet. Baseline position set on 2026-08-23.